American Journal of Public Health Research
ISSN (Print): 2327-669X ISSN (Online): 2327-6703 Website: Editor-in-chief: Jing Sun
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Special Issue "Tuberculosis and Respiratory Diseases Among Silica and Other Dust Exposed Workers"

A special issue of American Journal of Public Health Research (ISSN 2327-6703).

Special Issue Editors

Chief Guest Editor

Dr. Attapon Cheepsattayakorn
Department of Disease Control, Ministry of Public Health, Thailand

Guest Editor

Dr. Ruangrong Cheepsattayakorn
Department of Pathology, Faculty of Medicine, Chiang Mai University, Chiang Mai, Thailand

Special Issue Information

Airborne dusts are well known aerosols that associate with classical widespread occupational lung diseases, such as occupational asthma, chronic obstructive pulmonary disease (COPD), pneumoconiosis (such as silicosis, etc.), etc. There are several types of dust identified in the work environment, for examples : metallic dusts (lead, beryllium, nickel, cadmium dusts), mineral dusts (free crystalline silica, cement, and coal dusts), organic and vegetable dusts (cotton, wood, tea, pollen, and flour), biohazards (moulds and spores, and viable particles), and other chemical dusts (pesticides and several bulk chemicals). The commonest type of occupational dust particles includes silica, asbestos, and coal mine dusts. The main respiratory diseases associated with occupational dust particles include silicosis (caused by inhaling silica dust, usually associated with tuberculosis), asbestosis (caused by inhaling asbestos, usually associated with malignant pleural mesothelioma)), talc pneumoconiosis (caused by inhaling talc dust), berylliosis (caused by inhaling beryllium), siderosis (caused by inhaling iron oxide), metallic pneumoconiosis (caused by inhaling cobalt, barium, tungsten, tin dust), popcorn pneumoconiosis (caused by inhaling fumes produced when manufacturing microwave popcorn), kaolin pneumoconiosis (caused by inhaling China clay), and coal worker’ pneumoconiosis (CWP) (caused by inhaling coal dust).

Silica or silicon dioxide (SiO2) comprise around 25 % of known minerals, nearly 40 % of the common minerals, and more than 90 % of the earth’ s crust. Silica occurs in three forms : crystalline, microcrystalline (or cryptocrystalline) and amorphous (non-crystalline). Occupational exposure to crystalline silica can occur in any workplace, such as mining, mineral processing, slate working, quarrying, foundry work, stone crushing and dressing, brick and tile making, construction work, building restoration, tunneling, pottery and ceramic industries, and work with brick, concrete, stone, and some insulation boards. Silica-associated diseases remain an important public health problems among the low- and middle-income countries in the twenty-first century due to crystalline silica being one of a handful of toxins that causes several deadly diseases, such as pulmonary tuberculosis, lung cancer, silicosis, COPD, pulmonary fibronodular disease, renal diseases, and autoimmune diseases.

Exposure to silica for a long time, without silicosis may predispose persons to tuberculosis. The tuberculosis risk increases with the severity of silicosis that is considerably higher in patients with acute and accelerated silicosis. Cigarette smoking cessation program is important due to the interaction between silica exposure and cigarette smoking in the development of tuberculosis.

Occupational exposure to dust particles can occur anywhere, but it is particularly prevalent in the low- and middle-income countries. The aims and scope of this special issue are to share the knowledge and experiences in preventing, controlling including possible treating these deadly diseases among the workers around the world.

Special Issue Topics

About the issue

The topics to be covered by this special issue include, but are not limited to:

  • • Tuberculosis and Silica Exposure
  • • Silicosis
  • • Asbestosis
  • • Talc Pneumoconiosis
  • • Metallic Pneumoconiosis
  • • Berylliosis
  • • Siderosis
  • • Popcorn Pneumoconiosis
  • • Kaolin Pneumoconiosis
  • • Occupational COPD
  • • Occupational Asthma

Import Dates & Submit

Important dates

Submission Deadline: February 28, 2018
Notification of Acceptance: April 01, 2018
Final Version Due: May 01, 2018
Special Issue Publishing Date: July 01, 2018

Submit your article now

Manuscripts should be submitted as an attached file to an e-mail directed to the Chief Guest Editor, Dr. Attapon Cheepsattayakorn at the address: <>