American Journal of Public Health Research
ISSN (Print): 2327-669X ISSN (Online): 2327-6703 Website: http://www.sciepub.com/journal/ajphr Editor-in-chief: Apply for this position
Open Access
Journal Browser
Go
American Journal of Public Health Research. 2018, 6(2), 121-124
DOI: 10.12691/ajphr-6-2-16
Open AccessSpecial Issue

Asthma in Workers: An Overview

Watcharaphat Maneechaeye1, , Wat Mitthamsiri2, Atik Sangasapaviliya2 and Panitan Pradubpongsa2

1Department of Out-Patient, Phramongkutklao Hospital, Bangkok, Thailand

2Department of Medicine, Division of Allergy and Clinical Immunology, Phramongkutklao Hospital, Bangkok, Thailand

Pub. Date: March 22, 2018

Cite this paper:
Watcharaphat Maneechaeye, Wat Mitthamsiri, Atik Sangasapaviliya and Panitan Pradubpongsa. Asthma in Workers: An Overview. American Journal of Public Health Research. 2018; 6(2):121-124. doi: 10.12691/ajphr-6-2-16

Abstract

Asthma is a respiratory disease commonly found in general medical practice and in various fields of study. Furthermore, asthma may be found in every age range. Twenty five percent of adult-onset asthma is occupational asthma. Allergens that cause occupational asthma are commonly found in work place such as animal proteins, plant proteins, metal transition, chemical substances, etc. The guideline on medical practice of the American College of Chest Physicians 2008 divides occupational asthma into two categories: sensitizer-induced occupational asthma and irritant-induced occupational asthma. Pathogenesis of disease are based on Immunologically mediated with participation of specific IgE, Immunologically mediated without evidence of participation of IgE and Non-immunologic. The diagnosis of occupational asthma in terms of symptoms and signs does not differ from the diagnosis of general asthma. The difference is that the diagnosis of occupational asthma must be reassured that the exposure to allergens involves a contact to irritating substance within workplace. Cure by medicine for occupational asthma is not different from that for general asthma. The best treatment would be to avoid allergens in the workplace. The preventive measures may be erected.

Keywords:
asthma workers occupational

Creative CommonsThis work is licensed under a Creative Commons Attribution 4.0 International License. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/

References:

[1]  Balmes, J., Becklake, M., Blanc, P., Henneberger, P., Kreiss, K., Mapp, C., “American Thoracic Society Statement: occupational contribution to the burden of airway disease”, in Environmental and Occupational Health Assembly, American Thoracic Society.. Am J Respir Crit Care Med. 2003, 167: 787-97.
 
[2]  Tarlo, SM., Balmes, J., Balkissoon, R., Beach, J., Beckett, W., Bernstein, D., “Diagnosis and management of work-related asthma”, in American College of Chest Physiciansconsensus statement, Chest 2008, 134(3Suppl): 1S-41S.
 
[3]  Bernstein, IL., Bernstein, D.I., Chan-Yeung, M., Malo, J.L., Definition and classification of asthma. In: Asthmain the workplace, 3rd ed. Bernstein, IL, Chan-Yeung, M, Malo, JL, Berstein, DI, (Eds), Francis & Taylor, New York, 2006, 1.
 
[4]  Rioux, J.P., Malo, J.L., L’Archevêque, J., Rabhi, K., Labrecque, M., “Workplace-specific challenges as a contribution to the diagnosis of occupational asthma”, Eur Respir J, 2008, 32, 997-1003.
 
[5]  Mapp, C.E., Boschetto, P., Maestrelli, P., Fabbri, L.M., “Occupational asthma”, Am J Respir Crit Care Med, 2005, 172, 280-305.
 
[6]  Maestrelli, P., Boschetto, P., Fabbri, L.M., Mapp, C.E., “Mechanisms of occupational asthma”, J Allergy Clin Immunol, 2009, 123, 531.
 
[7]  Malo, J.L., Lemiere, C., Gautrin, D., Labrecque, M., “Occupational asthma”, Curr OpinPulm Med, 2004, 10, 57-61.
 
[8]  Bardana, E.J. Jr., “Occupational asthma”, J Allergy Clin Immunol, 2008, 121, S408-11.
 
[9]  Agius, R.M., “Why are some low-molecular-weight agents asthmagenic”, Occup Med, 2000, 15, 369.
 
[10]  Jarvis, J., Seed, M.J., Elton, R., “Relationship between chemical structure and the occupational asthma hazard of low molecular weight organic compounds” Occup Environ Med, 62, 243, 2005.
 
[11]  Bentley, A.M., Maestrelli, P., Fabbri, L.M., “Immunochemistry of the bronchial mucosa in occupational, intrinsic and extrinsic asthma”, J Allergy Clin Immunol, 1991, 87, 246.
 
[12]  Frew, A.J., Chan, H., Lam, S., Chan-Yeung, M., “Bronchial inflammation in occupational asthma due to western red cedar” Am J Respir Crit Care Med, 1995, 151, 340.
 
[13]  Gautrin, D., Bernstein, I.L., Brooks, S.M., Henneberger, P.K., Reactive airways dysfunction syndrome and irritant-induced asthma In: Bernstein IL, Chan-Yeung M, MaloJ-L, Bernstein DI, editors. Asthma in the workplace. 3rd ed. Francis & Taylor, New York, 2006, 581-630.
 
[14]  Beach J, Rowe BH, Blitz S, et al. “Diagnosis and Management of Work-Related Asthma:Summary”, Evidence Report/Technology Assessment, Number 129. AHRQ Publication Number 06-E003-1, October 2005. [Online]. Available: http://www.ahrq.gov/clinic/epcsums/asthworksum.htm/. [Accessed Jul. 2, 2017].