American Journal of Public Health Research
ISSN (Print): 2327-669X ISSN (Online): 2327-6703 Website: Editor-in-chief: Apply for this position
Open Access
Journal Browser
American Journal of Public Health Research. 2015, 3(5A), 99-101
DOI: 10.12691/ajphr-3-5A-21
Open AccessResearch Article

Dyselectrolytemia and Leucocytosis in Traumatic Brain Injury Patients

BalGopal Karmacharya1, Sarthak Nepal2, Madhav Acharya3, Silpa Sharma2 and Pranita Neupane2

1Consultant, Neurosurgery Unit, Manipal Teaching Hospital, Nepal

2Intern, Manipal Teaching Hospital, Nepal

3Medical Officer, Manipal Teaching Hospital, Nepal

Pub. Date: October 28, 2015
(This article belongs to the Special Issue Health Scenario 2015; Millennium Development Goals)

Cite this paper:
BalGopal Karmacharya, Sarthak Nepal, Madhav Acharya, Silpa Sharma and Pranita Neupane. Dyselectrolytemia and Leucocytosis in Traumatic Brain Injury Patients. American Journal of Public Health Research. 2015; 3(5A):99-101. doi: 10.12691/ajphr-3-5A-21


Traumatic brain injuries are common. These constitute an important cause of morbidity and mortality in people of all age groups. Electrolyte disturbance and leucocytosis are commonly seen in patients with traumatic brain injuries. This study was done to find out the frequency of electrolyte disturbances and leucocytosis in patients with traumatic brain injuries. This was a descriptive study of all patients who were brought to the emergency room of Manipal Teaching Hospital. Demographic parameters, cause of injury, CT scan findings, severity of injury were noted in a proforma. Serum sodium and potassium level and total count was measured in blood samples taken from all patients in the emergency. There were 85 patients included in the study. Abnormal sodium level was found in 36.5% and abnormal potassium level was found in 10.6%. Similarly almost two thirds of patients had leucocytosis. Electrolyte disturbances should be measured and meticulously treated in patients with traumatic brain injuries.

traumatic brain injury

Creative CommonsThis work is licensed under a Creative Commons Attribution 4.0 International License. To view a copy of this license, visit


[1]  Costa KN, Nakamura HM, Cruz LR, Miranda LS, Santos-Neto RC et al. Hyponatremia and brain injury: absence of alterations of serum brain natriuretic peptide and vasopressin. Arq Neuropsiquiatr. 2009. Dec ; 67(4):1037-44.
[2]  Isotani E, Suzuki R, Tomita K. Alterations in plasma concentrations of natriuretic peptides and antidiuretic hormone after subarachnoid hemorrhage. Stroke. 25 Nov; (11):2198-203.
[3]  Lath R. Hyponatremia in neurological diseases in ICU. Indian J Crit Care Med. 9:47-51.
[4]  Gürkanlar D, Lakadamyali H, Ergun T, Yilmaz C, Yücel et al. Predictive value of leucocytosis in head trauma. Turk Neurosurg. 2009 Jul; 19(3):211-5.
[5]  Ishizaki T, Momota H, Kuwahara K, Tanooka A, Morimoto S. A. case of symptomatic traumatic cerebral vasospasm associated with hyponatremia. No Shinkei Geka 1999 Nov; 27: 1031-6. [Articlein Japanese].
[6]  Choon Hong Kan, Mohd Saffari, Teik Hooi Khoo. Prognostic Factors of Severe Traumatic Brain Injury Outcome in Children Aged 2-16 Years at a Major Neurosurgical Referral Centre. Malays J Med Sci. 2009 Oct;16(4): 25-33.
[7]  Aakash Bodhit et al. Hyperglycemia And Leukocytosis As Predictors Of Abnormal Head CT Following Concussion. Neurology April 8, 2014 vol. 82 no. 10 Supplement P5. 301.
[8]  Cerda-Esteve M, Ruiz-Gonzalez A, Gudelis M, Goday A, Trujillano J et al. Incidence of hyponatremia and its causes in neurological patients. EndocrinolNutr. 2010; 57(5): 182-6.
[9]  Upadhyaya A, Jaber BL, Madias NE. Incidence and prevalence of hyponatremia. Am J Med. 2006Jul; 119(7 Suppl 1):S30-5.
[10]  Usha S Adiga, Vickneshwaran V, Sanat Kumar Sen. Electrolyte derangements in traumatic brain injury. Basic Research Journal of Medicine and Clinical Sciences ISSN 2315-6864 Vol. 1(2) p. 15-18 September 2012.