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Scalbert, A., Williamson, G., “Dietary intake and bioavailability of polyphenols.” J. Nutr, 130. 2073S-2085S. 2000.

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Article

Defensive Effect of Quercetin against Tumor Necrosis Factor α-induced Endoplasmic Reticulum Stress and Hepatic Insulin Resistance in HepG2 Cells

1Division of Food and Nutrition, Chonnam National University, Gwangju, South Korea

2Department of Medical Nutrition, Kyung Hee University, Yongin, South Korea


Journal of Food and Nutrition Research. 2018, Vol. 6 No. 8, 518-524
DOI: 10.12691/jfnr-6-8-6
Copyright © 2018 Science and Education Publishing

Cite this paper:
Jeongjin Park, Woojin Jun, Jeongmin Lee, Ok-Kyung Kim. Defensive Effect of Quercetin against Tumor Necrosis Factor α-induced Endoplasmic Reticulum Stress and Hepatic Insulin Resistance in HepG2 Cells. Journal of Food and Nutrition Research. 2018; 6(8):518-524. doi: 10.12691/jfnr-6-8-6.

Correspondence to: Ok-Kyung  Kim, Division of Food and Nutrition, Chonnam National University, Gwangju, South Korea. Email: jlee2007@khu.ac.kr, okkyung85@gmail.com

Abstract

In order to examine the hypothesis that the treatment of TNF-α can impose inflammation and endoplasmic reticulum (ER) stress in hepatic cells, and that quercetin has a defensive effect against TNF-α-induced ER stress and insulin resistance, we evaluated the effect of quercetin (3 µg/mL and 5 µg/mL) in the TNF-α-induced HepG2 cells. The TNF-α-stimulated control group caused a marked increase in the activation of inflammation and ER stress response. Quercetin, however, caused the interruption of TNF-α-induced inflammation and ER stress. In addition, the treatment of quercetin resulted in significant decreases in serine phosphorylation of IRS-1, phosphorylation of JNK, and the expression of gluconeogenic genes compared with the TNF-α-stimulated control group. In conclusion, we suggest that quercetin can protect hepatic insulin resistance by exerting a protective effect against the ER stress and inflammation induced by TNF-α.

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