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Szaflarska-Poplawska, A., Siomek, A., Czerwionka-Szaflarska, M., Gackowski, D., Rozalski, R., Guz, J., Szpila, A., Zarakowska, E., Olinski, R. Oxidatively damaged DNA/oxidative stress in children with celiac disease. Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology, 19 (8):1960-5. 2010.

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Article

Coeliac Disease: From Triggering Factors to Treatment

1Research Group for Pediatrics and Nephrology, Hungarian Academy of Sciences and Semmelweis University, Budapest, Hungary

2First Department of Pediatrics, Semmelweis University, Budapest, Hungary

3“Momentum” Diabetes Research Group, Hungarian Academy of Sciences and Semmelweis University, Budapest, Hungary


International Journal of Celiac Disease. 2013, Vol. 1 No. 1, 9-13
DOI: 10.12691/ijcd-1-1-5
Copyright © 2013 Science and Education Publishing

Cite this paper:
Erna Sziksz, Péter Vörös, Gábor Veres, Andrea Fekete, Ádám Vannay. Coeliac Disease: From Triggering Factors to Treatment. International Journal of Celiac Disease. 2013; 1(1):9-13. doi: 10.12691/ijcd-1-1-5.

Correspondence to: Erna  Sziksz, Research Group for Pediatrics and Nephrology, Hungarian Academy of Sciences and Semmelweis University, Budapest, Hungary. Email: szikszerna@gmail.com

Abstract

Coeliac disease (CD) or gluten sensitive enteropathy is one of the most common inflammatory diseases of the small intestine with estimated prevalence of 1% in the population. Its incidence is increasing and seems to be higher than expected in the pediatric population associated with unfavorable impact on the quality of life. The aim of the present review is to highlight the main triggering factors leading to the development of CD and its pathomechanism with a special outlook to the recent therapeutic approaches.

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