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Simondi D, Ribaldone DG, Bonagura GA, Foi S, Sapone N, Garavagno M, Villanacci V, Bernardi D, Pellicano R, Rizzetto M, Astegiano M. Helicobacter pylori in celiac disease and in duodenal intraepithelial lymphocytosis: Active protagonist or innocent bystander?. Clinics and research in hepatology and gastroenterology. 2015 Dec 31; 39(6): 740-5.

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Article

Triggering Agents for Transient Celiac Disease

1Department of Pediatrics, University Hospital of Setif, Setif 1 University, Algeria

2Genetic, Cardiovascular & Nutritional Diseases Laboratory, Setif-1 University, Algeria

3Biochemistry Laboratory, University Hospital of Setif, Setif 1 University, Algeria


International Journal of Celiac Disease. 2017, Vol. 5 No. 3, 127-128
DOI: 10.12691/ijcd-5-3-5
Copyright © 2017 Science and Education Publishing

Cite this paper:
Hakim Rahmoune, Nada Boutrid, Mounira Amrane, Belkacem Bioud. Triggering Agents for Transient Celiac Disease. International Journal of Celiac Disease. 2017; 5(3):127-128. doi: 10.12691/ijcd-5-3-5.

Correspondence to: Hakim  Rahmoune, Department of Pediatrics, University Hospital of Setif, Setif 1 University, Algeria. Email: rahmounehakim@gmail.com

Abstract

Celiac disease is a genetically determined gluten-sensitive auto-immune condition, mainly an enteropathy resulting in nutrient malabsorption. Its intestinal damages, with lymphocytic gastroduodenitis, can also be due to a myriad of gastro-intestinal infections that act as true "triggering agents". We present two pediatric patients with infectious associations leading to a transient positive celiac disease serology, with various agents and different genetic suscpetibility.

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