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Saberi S, Schmidt A, Eybpoosh S, Esmaili M, Talebkhan Y, Mohajerani N et al. Helicobacter pylori Strains from Duodenal Ulcer Patients Exhibit Mixed babA/B Genotypes with Low Levels of BabA Adhesin and Lewis b Binding. Dig Dis Sci. 2016 Jun 18. [Epub ahead of print].

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Article

The Gut-stomach Axis: Helicobacter pylori and Celiac Disease

1B. Rappaport School of Medicine, Technion-Israel Institute of Technology, Haifa, 34362, Israel

2AESKU.KIPP Institute, Wendelsheim 55234, Germany


International Journal of Celiac Disease. 2016, Vol. 4 No. 3, 77-79
DOI: 10.12691/ijcd-4-3-10
Copyright © 2016 Science and Education Publishing

Cite this paper:
Aaron Lerner, Torsten Matthias. The Gut-stomach Axis: Helicobacter pylori and Celiac Disease. International Journal of Celiac Disease. 2016; 4(3):77-79. doi: 10.12691/ijcd-4-3-10.

Correspondence to: Aaron  Lerner, B. Rappaport School of Medicine, Technion-Israel Institute of Technology, Haifa, 34362, Israel. Email: aaronlerner1948@gmail.com

Abstract

Abnormalities in the gastric and duodenal eco-systems have been proposed as contributing to the pathophysiology of celiac disease (CD) and Helicobacter pylori (Hp) infection, respectively. Lymphocytic gastritis and lymphocytic duodenitis cohabit in Hp and celiac disease and also in Hp- or non-CD respective conditions. The present editorial discusses those entities, concentrating on the pathophysiological pathways, highlighting the Hp-CD relationship in the contest of the gut-stomach axis.

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