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<!DOCTYPE ArticleSet PUBLIC "-//NLM//DTD PubMed 2.0//EN" "http://www.ncbi.nlm.nih.gov:80/entrez/query/static/PubMed.dtd"[]>
<ArticleSet>
  <Article>
    <Journal>
      <PublisherName>Science and Education Publishing</PublisherName>
      <JournalTitle>Journal of Cancer Research and Treatment</JournalTitle>
      <Volume>2</Volume>
      <Issue>1</Issue>
      <PubDate PubStatus="epublish">
        <Year>2014</Year>
        <Month>02</Month>
        <Day>21</Day>
      </PubDate>
    </Journal>
    <ArticleTitle>Murine Models of Cancer</ArticleTitle>
    <FirstPage>16</FirstPage>
    <LastPage>18</LastPage>
    <Language>EN</Language>
    <AuthorList>
      <Author>
        <FirstName>Ahmed M</FirstName>
        <LastName>Kabel</LastName>
        <Affiliation>Department of Pharmacology, Faculty of Medicine, Tanta University, Egypt</Affiliation>
      </Author>
      <Author>
        <FirstName>Maaly A Abd</FirstName>
        <LastName>Elmaaboud</LastName>
      </Author>
    </AuthorList>
    <ArticleIdList>
      <ArticleId IdType="pii">JCRT2014214</ArticleId>
      <ArticleId IdType="doi">10.12691/jcrt-2-1-4</ArticleId>
    </ArticleIdList>
    <History>
      <PubDate PubStatus="received">
        <Year>2013</Year>
        <Month>10</Month>
        <Day>12</Day>
      </PubDate>
      <PubDate PubStatus="revised">
        <Year>2014</Year>
        <Month>12</Month>
        <Day>29</Day>
      </PubDate>
      <PubDate PubStatus="accepted">
        <Year>2014</Year>
        <Month>02</Month>
        <Day>21</Day>
      </PubDate>
    </History>
    <Abstract>Cancer is a class of diseases in which a group of cells displays uncontrolled growth and invasion that destroys adjacent tissues, and sometimes send metastasis to other locations in the body. When one is interested in cancer research, mice will be the preferred animal. Murine models of cancer include transplantable models, spontaneous and autochthonous models, human tumor xenografts, orthotopic models, models of metastasis and transgenic tumor models.</Abstract>
  </Article>
</ArticleSet>