American Journal of Medical Case Reports
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American Journal of Medical Case Reports. 2019, 7(7), 151-157
DOI: 10.12691/ajmcr-7-7-9
Open AccessCase Report

Right Ventricular Infarction Associated with Pulmonary Embolism

Mohammed Al-Sadawi1, Rishard Abdul1 and Samy I. McFarlane1,

1Department of Internal Medicine, State University of New York: Downstate Medical Center, Brooklyn, New York, United States- 11203

Pub. Date: July 05, 2019

Cite this paper:
Mohammed Al-Sadawi, Rishard Abdul and Samy I. McFarlane. Right Ventricular Infarction Associated with Pulmonary Embolism. American Journal of Medical Case Reports. 2019; 7(7):151-157. doi: 10.12691/ajmcr-7-7-9


Background: Since the introduction of heparin as part of the management of acute coronary syndrome, the occurrence of pulmonary embolism (PE) as a complication of myocardial infarction (MI) have somewhat been unheard of. Given this rarity, its recognition is now a formidable challenge. Case Description: We present a case of a previously well 56-year-old male who presented with typical chest pain radiating to the left arm and dizziness. Initial vital signs revealed a blood pressure of 95/65 mmHg and his heart rate was 42 bpm. Physical exam revealed a middle-aged male in no acute distress with cool extremities, normal heart sounds, no murmurs and flat neck veins. His chest was clear to auscultation, abdomen benign and clinically, he was euvolemic. His first electrocardiogram (EKG) was significant for bradycardia with heart block and junctional escape rhythm and ST depressions in the anteroseptal leads. Subsequent right sided EKG showed sinus bradycardia with ST segment elevations in leads II, III and aVF. Troponin I was 0.95 ng/L [normal <0.04 ng/L] and initial transthoracic echo (TTE) was unremarkable with an ejection fraction (EF) of 55-60%. He was given loading doses of aspirin and clopidogrel, heparin drip initiated, and he proceeded to cardiac catheterization which revealed a 60 % stenotic lesion of the mid LAD and a large filling defect with 100% stenosis of the mid RCA consistent with thrombus. Post procedure TTE revealed an EF of 40%, septal and posterior hypokinesis, right ventricular regional wall motion abnormality of the basal and mid free wall with apical hypercontractility (McConnell's sign) suggestive of PE. CT pulmonary angiography revealed bilateral pulmonary emboli and anticoagulation therapy was initiated. Conclusion: This case illustrates that pulmonary embolism is a potential complication of RV infarction and its early identification is critical for implementation of anticoagulation therapy for this potentially fatal condition if left undiagnosed and untreated.

right ventricular infarction pulmonary embolism coronary intervention

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